RAYNAUD’S PHENOMENON
Raynaud’s phenomenon involves bilateral paroxysmal ischemia of fingers or toes, usually precipitated by cold and relieved by heat. The entire hand or foot is usually not affected. Raynaud’s phenomenon may be secondary to a variety of underlying diseases, including arterial occlusive diseases (e.g., arteriosclerosis obliterans or thromboangiitis obliterans), connective tissue diseases (e.g., scleroderma, rheumatoid disease, systemic lupus erythematosus, or polyarteritis nodosa), trauma (e.g., air hammer operators or typists), various neurologic diseases (e.g., multiple sclerosis or peripheral neuropathy), ingestion of certain drugs (e.g., ergotamine, methysergide, or beta-adrenergic blocking drugs), nerve compression syndromes (e.g., thoracic outlet or carpal tunnel syndrome), hematologic abnormalities (e.g., cryoglobulinemia or cold agglutinins), and late after-cold injuries (e.g., frostbite or trench foot). The phenomenon probably occurs secondary to abnormalities of the sympathetic nerves, abnormal reactivity of the blood vessels, and/or sludging of blood cells in the capillaries. If no underlying disease can be found, the patient is said to have Raynaud’s disease. Raynaud’s disease affects women more frequently than men, most commonly between the ages of puberty and 40.
At the time of an attack, the affected digit initially turns white and subsequently becomes cyanotic. Upon resumption of circulation, it may appear hyperemic because of vasodilation. These changes are usually precipitated by exposure to cold but may also occur with emotional upset. In Raynaud’s disease the changes are usually bilateral. Between attacks, normal pulses are present, and the digits usually appear normal. In more severe cases “trophic” changes in the fingers or toes may occur consisting of smooth, shiny, tightly stretched skin over the digits (sclerodactyly) and nail changes. Small painful areas of gangrene may appear on the tips of digits and leave scars upon healing. Amputations of digits in patients with Raynaud’s disease are rare.
Mild cases should be treated by limitation of cold exposure. Smoking should be avoided. In more severe cases, vasodilating agents such as calcium-channel blocking agents may be used. Some patients may respond to regional sympathectomy. In Raynaud’s phenomenon treatment should be aimed at the underlying cause.
- HEART DISEASE AND PREGNANCY
- THE AIRWAY STRUCTURE
- VENTILATION
- GAS TRANSFER
- NONPULMONARY FACTORS
- ENVIRONMENTAL DAMAGE OF THE EXTREMITIES
- THROMBOANGIITIS OBLITERANS
- THE BLOOD VESSELS STRUCTURE
- GENERAL PRINCIPLES OF CARDIAC SURGERY
- RAYNAUD'S PHENOMENON
- GENERAL SURGERY IN THE PATIENT WITH HEART DISEASE
- PERIPHERAL ANEURYSMS AMD FISTULAE
- PULMONARY GAS EXCHANGE
- ARTERIOSCLEROSIS OBLITERANS
- MISCELLANEOUS AORTIC DISEASE
- ARTERIAL TRAUMA
- PERFUSION
- PERIPHERAL VENOUS DISEASE
- NONRESPIRATORY FUNCTIONS OF THE LUNG